Nap1 stimulates homologous recombination by RAD51 and RAD54 in higher-ordered chromatin containing histone H1

نویسندگان

  • Shinichi Machida
  • Motoki Takaku
  • Masae Ikura
  • Jiying Sun
  • Hidekazu Suzuki
  • Wataru Kobayashi
  • Aiko Kinomura
  • Akihisa Osakabe
  • Hiroaki Tachiwana
  • Yasunori Horikoshi
  • Atsuhiko Fukuto
  • Ryo Matsuda
  • Kiyoe Ura
  • Satoshi Tashiro
  • Tsuyoshi Ikura
  • Hitoshi Kurumizaka
چکیده

Homologous recombination plays essential roles in mitotic DNA double strand break (DSB) repair and meiotic genetic recombination. In eukaryotes, RAD51 promotes the central homologous-pairing step during homologous recombination, but is not sufficient to overcome the reaction barrier imposed by nucleosomes. RAD54, a member of the ATP-dependent nucleosome remodeling factor family, is required to promote the RAD51-mediated homologous pairing in nucleosomal DNA. In higher eukaryotes, most nucleosomes form higher-ordered chromatin containing the linker histone H1. However, the mechanism by which RAD51/RAD54-mediated homologous pairing occurs in higher-ordered chromatin has not been elucidated. In this study, we found that a histone chaperone, Nap1, accumulates on DSB sites in human cells, and DSB repair is substantially decreased in Nap1-knockdown cells. We determined that Nap1 binds to RAD54, enhances the RAD54-mediated nucleosome remodeling by evicting histone H1, and eventually stimulates the RAD51-mediated homologous pairing in higher-ordered chromatin containing histone H1.

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عنوان ژورنال:

دوره 4  شماره 

صفحات  -

تاریخ انتشار 2014